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Concurrently, a significant increase in NADH/NAD + ratio ( P = 0.0002) was observed (Figure 6C), suggesting that the observed metabolic disruptions were a result of oxidative stress, potentially as a compensatory mechanism to sustain energy production and redox homeostasis in the absence of GR

For instance, vitamin K2 (VK2) mitigates cartilage injury in an in vivo rat OA model induced by ACLT and in an in vitro model of oxidative damage in chondrocytes caused by tert-butyl hydroperoxide (TBHP)

Lipid profile alterations and biomarker identification in type 1 diabetes mellitus patients under glycemic control

doi: 10.1123/jpah.6.1.43

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