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Description
Mon1a was identified as a modifier gene of cellular iron metabolism via trafficking of FPN to the surface of iron-recycling macrophages [29]
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Although N1m-mRNA is less detectable by the toll-like receptors (TLR) 7 and 8 and considered a safer modality, it requires continued surveillance to address uncertainties around maternal-fetal impacts
PubMed CrossRef Anton RF, Myrick H, Wright T.M, et al
