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L., Malka, O., Hefetz, A., Tarpy, D
O-GlcNAcylation-dependent upregulation of HO1 triggers ammonia-induced oxidative stress and senescence in hepatic encephalopathy [J]
Baggio C
In the context of MASLD, glucagon receptor agonism may complement GLP-1 receptor agonism by directly stimulating hepatic fatty acid oxidation and reducing lipogenesis, leading to decreased hepatic steatosis
Below, we summarize novel therapeutic targets and strategies with clinical application potential to provide more options for disease treatment (Table 1)
