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Some preclinical data suggest additional anti-inflammatory or Kupffer-cellmediated effects, but the dominant, best-supported mechanism is metabolic: less substrate delivered to a less insulin-resistant liver

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Tetrahydropyrroloquinolinone type dual inhibitors of aromatase/aldosterone synthase as a novel strategy for breast cancer patients with elevated cardiovascular risks

American Journal of Clinical Nutrition 1994;60(1):211
