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10.1007/978-3-319-99375-1_45 [DOI] [Google Scholar] 288.Pietrobon AJ, Teixeira FME, Sato MN
This section and Table 4 include different potential targets specific to different ROS-mediated cellular processes or pathways, as well as potential therapeutic strategies for mitigating cancer
MET amplification in metastatic colorectal cancer: an acquired response to EGFR inhibition, not a de novo phenomenon
Riboflavin is also essential for the metabolism of homocysteine, as a cofactor for methylenetetrahydrofolate reductase (MTHFR) and methionine synthase reductase (MTRR) (11)
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