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Evidence suggests apolipoprotein A-V (apoA-V) may have an inhibitory effect on this process as apoA-V modulates VLDL TG mobilization as well as secretion [40]

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Furthermore this imbalance is central to pathologies like cancer, diabetes, and sepsis, highlighting monocytes as key therapeutic targets for restoring immune homeostasis. Treatment The authors reported that the viral persistence, immune activation, mitochondrial dysfunction, and metabolic derangements seen all represented potential treatment targets

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Safford, MM