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increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

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increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

Moreover, GSTs from the delta and epsilon classes in D

increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

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increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

[PubMed: 15060092] 845

increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

The extrinsic pathway is initiated by members of the death receptor family

increased glutathione in acute myeloid leukemia ABCC1 and metabolism limit the efficacy of BCL-2 inhibitors APR-246 induces early cell death

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