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doi: 10.1038/s41422020-03327 33 ChenLGuoYHuCLamPKSLamJCWZhouB

Although early NAC treatment is beneficial in limiting the injury, continuous NAC treatment well beyond the injury phase can delay regeneration [72] mainly through impairment of mitochondrial biogenesis, an adaptive response whose role is to maintain or restore homeostasis following mitochondrial dysfunction [71]

Importantly, an accumulation of reactive oxygen species (ROS) can lead to the additional production of ROS by many different mechanisms

This paper is a large meta-analysis of GWAS that describes the genetic variants that alter the risk for Parkinson disease